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133 lab tests, with examination and deep analysis, allows the patient full access to all supplemental optional testing, Therapure Nutraceuticals medicines, and personalized natural health services at BSI.
BSI certified medical staff first review the extensive on-line questionnaire the patient is asked to complete (next step by pressing the Health History Questionnaire and Booking button). Once accepted for therapy at BSI, the patient is then scheduled for two appointments.
The first appointment begins with review and clarification of answers given by the patient from the on-line questionnaire.
This is followed by physical examination that includes simple cardio-pulmonary testing, aural heart monitoring, blood pressure check, temperature-circulation quality check, oxygen saturation, abdominal palpitation if needed, BMI, and viewing of any complaints regarding skin, organs, etc.
Blood and urine samples are requested, and are immediately processed at the BSI onsite labs. Two drops of this blood are placed on a microscope slide, for preliminary diagnoses preformed within full view of the patient on high-resolution monitor.
The BSI technician will search for anomalies and their meanings in the blood (many of which do not present in conventional blood and urine lab testing), such as fungus, bacteria, parasites, damage to red cells, quality and type of white blood cells (immunities), serum (clear blood) observation, and vital force (the ability of blood to continue flowing outside of the body). These observations work in consort with blood laboratory testing to further clarify modern problems, such as antibiotic resistance, fungal infections, and more.
All New Member Patients are requested to complete the Health History Questionnaire, with Full Basic Testing, as described below.
Based in part on the Health History Questionnaire you would complete before arriving at our clinic. All new patients are accepted via this extensive form.
Which is also used to to further refine test results and better determine possible prescription for herbal medicines and therapies at BSI.
Blood pressure, oxygen percentage, heart rate and stability, body temperature, BMI, BMR, BFC, weight, respiratory function and rating, growths under and on skin, abdominal palpitation if needed, etc.
with comprehensive written explanation. Using state-of-art German technology. Click here for a complete listing of all tests an examinations included ....
Plus basic blood tests for kidney / urinary function
(as determined from white blood cell Differential Count, epithelial cell count, and other indices as read together) (does not include tumor antigen tests, which are optional)
(You watch on hi-res monitor) to verify and preview results from mechanized testing as listed above
STD Testing, Thyroid & Pituitary Testing, Lipid Profile, Tumor Marker Testing, Heavy Metals Analysis Testing, Male Hormone, Female Hormone.
Will be explained via return email, once your Health History Questionnaire has been completed and received at BSI, and you have been accepted as a patient. (We need to know what your needs are before we can determine time and costs for you.)
This allows you to receive our medicines and therapies at any time, and also to write to us for information regarding any health problems we are able to address at BSI. (BSI Premium medicines, therapies, and consultation are exclusively available to Member Patients.)
And a large variety of optional additional tests can be added during first consultation, or returning test results consultation, if requested by the Patient or the BSI Practitioner. Additional testing is only recommended on average to 1 in 20 patients.
Test results and prescription (if needed) are presented to the patient and explanations given in detail during the second visit, 2 – 3 business days later.
See below for a complete list of included tests.
Full Analysis of all 133 tests is documented on a 28 – 34 page report, complete with photos and combined meanings of the tests. This report is presented to the patient, explained page by page. A prescription is included at the end, if needed.
In live blood analysis, serum refers to the liquid portion of the blood that remains after the blood cells have been separated through clotting. It is a key focus in non-magnified blood observations because it provides valuable information about the biochemical state of the body. The serum contains proteins such as albumin and globulins, electrolytes like sodium and potassium, as well as metabolic waste products and hormones. Observing the serum in its natural state can help identify imbalances, nutrient deficiencies, or the presence of inflammatory markers.
Dehydration in live blood analysis is observed when the blood appears more concentrated than normal. In a non-magnified view, this can be seen as thicker plasma with blood cells that are more tightly packed together, often leading to a reduction in the volume of plasma compared to the cellular components. Dehydration can have serious implications for blood flow, as it reduces the blood’s ability to carry oxygen and nutrients to the cells. As the plasma becomes more viscous due to insufficient fluid, it can lead to sluggish circulation, increased stress on the heart, and reduced efficiency in waste elimination.
Clotting in live blood analysis refers to the formation of fibrin strands or blood clots, which occur when blood cells aggregate in an attempt to seal an injury or stop bleeding. Non-magnified blood observations can help identify clotting tendencies by looking for signs such as abnormal clumping of blood cells or the presence of fibrin strands in the serum. These clots may be indicative of underlying health issues like an inflammatory response, oxidative stress, or even blood coagulation disorders, including conditions such as thrombophilia or hypercoagulability. In some cases, excessive clotting can lead to poor circulation, increasing the risk of thrombosis, heart attack, or stroke.
Platelets or thrombocytes react to bleeding from blood vessel injury by clumping, thereby initiating a blood clot. Platelets have no cell nucleus. Platelets congregate around a wound creating a cap to stop blood flow out of the tissue (clotting). Platelets also contain cytokines and growth factors which can promote wound healing and regeneration of damaged tissues.
Platelets or thrombocytes react to bleeding from blood vessel injury by clumping, thereby initiating a blood clot. Platelets have no cell nucleus; they are fragments of cytoplasm derived from the megakaryocytes of the bone marrow or lung, which then enter circulation. Platelets congregate around a w ound creating a cap to stop blood flow out of the tissue (clotting). Platelets also contain cytokines and growth factors which can promote wound healing and regeneration of damaged tissues.
Infrequent acanthocytes are often encountered in hyposplenic conditions, but where they are very frequent this may indicate an uncommon or serious cause which needs to be communicated to clinicians. Light or severe liver disease causing coagulopathy and spur cell (acanthocytic) anaemia.
Agglutinates arise when antibodies attach to antigens on the membranes of adjacent red cells linking them together. The most common cause is “cold-reactive” IgM antibodies which do not cause overt symptoms. However, in some cases the effects may be clinically significant since antibodies may activate complement causing haemolysis, or the agglutinated cells can cause occlusion of small blood vessels in the cold (acrocyanosis). The clumped cells will sediment more rapidly leading to a raised erythrocyte sedimentation rate (ESR). Finally, the antibodies that cause cold agglutination may indicate an underlying malignancy (particularly lymphoma), or by acute infection.
Most commonly arises when blood cell production is stressed or abnormal, may be associated with dysfunction of enzymes involved in RNA breakdown (either congenital deficiency or drug induced).
Although most closely associated with classical sickle disease (HbSS), boatshaped cells are also seen in compound heterozygotes between HbS and other abnormal haemoglobins.
Ignore boat cells in areas of blood movement, only consider if blood is static.
Cabot rings are ring-like or figure-of-eight loop-shaped inclusions composed of microtubule remnants from the mitotic spindle, or possibly nuclear remnants or abnormal histones. Can indicate B-12 anemia and related diseases, megaloblastic anemia, myelodysplastic syndrome, and lead poisoning.
Strongly indicative of a “packed marrow”. May be the result of fibrosis (primary or secondary) or neoplasm (carcinoma or hematological neoplasm). May also arise where there is sustained or severe physiological increase in blood cell production (e.g. the expanded erythroid response to thalassemia). Less frequent tear drop forms may arise in other systemic disease. Ignore in fast-moving blood (vital force).
Iron deficiency or chronic disease. Multiple instances can indicate very severe hereditary pyropoikilocytosis.
Always remember that echinocytes may be an artefact of blood storage or of cells on the edge of a live mount slide (artefactual echinocytosis), so look at the condition of other cells on the film and determine whether the echinocytosis is patchy in distribution. Where genuine there will usually be a significant systemic disease present. This most frequently will be renal failure.
The presence of hypochromia indicates defective production of hemoglobin. Most cases result from iron deficiency or thalassemia – other typical features of these conditions should therefore be sought. Less frequently, hypochromia reflects defective iron utilization (e.g. chronic disease or sideroblastic anemia). The presence of hypochromia is not of itself an urgent problem unless there is severe anemia; however, it important to highlight the condition since clinicians may need to request further investigation to determine its cause.
Howell-Jolly Bodies most commonly arise when spleen is absent or spleen function is impaired (hyposplenia). Occasional Howell Jolly bodies may arise in physiological conditions.
Implies damage to hemoglobin within the red cell often accompanied by cellular dehydration and membrane damage; acute oxidative damage to red cells should be considered.
In some (although not all) cases, the pathological process may be life threatening particularly if they are associated with disseminated intravascular coagulation (DIC) or thrombotic thrombocytopenic purpura (TTP) knowledge of platelet count, clotting and additional morphological features such as fragments is essential.
Morphological evidence of any accompanying disease should actively be sought. Most frequently these causes are B12 or folate deficiency, myelodysplasia, or liver disease.
Myelodysplasia, iron deficiency, etc. S.E. Asian Ovalocytosis is a specific disorder that results from structural and functional defects of the band 3 protein causing ovalocytes with a stomatocytic appearance. May indicate previous malarial parasites.
Small numbers of Pappenheimer Bodies may be seen in normal blood, particularly within polychromatic cells. When they are present in large number look for hyposplenic features, or for pathological states that have iron-loading or aberrant iron metabolism.
These cells are formed when sickle hemoglobin (HbS) is present together with hemoglobin C (HbC) to form a compound heterozygote disorder (HbSC disease)
Fragmented cells are not found in normal blood. Sharp fragments may reflect “microangiopathic” damage – this form of fragmentation may therefore represent a medical emergency and should be reported immediately. More rounded fragments arise in significant dyserythropoiesis (such as severe myelodysplasia, membrane disorder or megaloblastic states), these are also important to diagnosis, but have a different origin.
Indicates that the cells express the mutated gene for sickle hemoglobin (HbS), either in homozygous form (HbSS) or as a compound with another abnormal beta hemoglobin form. The number of these abnormal cells should not necessarily be considered an indicator of severity, but increased numbers of abnormal cells and polychromasia (or nucleated red cells) often occur during sickle crises.
First exclude artefact (slow slide drying), then consider the number of these cells – occasional stomatocytes may arise in many conditions. However, when present in significant number stomatocytes may indicate significant acquired or inherited disease signifying one of a range of conditions (including liver disease, alcohol, or electrolyte imbalance). However, also consider a range of inherited conditions (see table) or look for the characteristic very large and oval-shaped often with a Y-shaped stoma that are seen in South East Asian Ovalocytosis.
Spherocytes are found in all hemolytic anemias to some degree. Hereditary spherocytosis and autoimmune hemolytic anemia are characterized by having only spherocytes. Where spherocytes are very frequent autoimmune hemolysis or hereditary spherocytosis should be considered.
Severe membrane defects (e.g. hereditary pyropoikilocytosis). Toxin induced membrane damage: particularly Clostridium perfringens. Infrequent microspherocytes may appear as part of a spectrum of cells in many conditions with erythrocyte damage (e.g. fragmentation) or fragile production (e.g. megaloblastic states).
The area of pallor contains a central accumulation of hemoglobin giving the appearance of a “target”. Look for macrocytosis that may imply liver disease; or if MCV is normal or low consider a hemoglobinopathy (HbC, D or E).
Neutrophils help heal damaged tissues and resolve infections. Neutrophil blood levels increase naturally in response to infections, injuries, and other types of stress. They may decrease in response to severe or chronic infections, drug treatments, and genetic conditions.
Lymphocytes help fight disease and infection. They are primarily involved in recognizing and responding to foreign substances, such as viruses and bacteria, with two main types: T cells, which destroy infected cells, and B cells, which produce antibodies to target pathogens.
Monocytes influence adaptive immune responses and exert tissue repair functions. Monocytes are mechanically active cells and migrate from blood to an inflammatory site to perform their functions. In general, monocytes and their macrophage and dendritic cell progeny serve three main functions in the immune system.
Normally transparent, it is this affinity that causes them to appear brick-red after staining. Eosinophils are responsible for combating multicellular parasites and certain infections in vertebrates. Along with mast cells and basophils, they also control mechanisms associated with allergy and asthma.
Basophils release enzymes to improve blood flow and prevent blood clots. Basophils function to defend your body against:
Stem cells will appear like red cells, but do not clot and display a large white center.
Hematopoietic stem cells (HSC) emanate from bone marrow and can produce all the cells that function in the blood. Stem cells also can become brain cells, heart muscle cells, bone cells or other cell types.
Hematopoietic stem cells (HSC) circulate under steady state conditions in peripheral blood to
i) to maintain a stem cell pool in remote bone marrow locations in the body and
ii) to “patrol” peripheral tissues and organs and, when needed, to respond to organ injuries and infections. The number of these cells increases in stress situations related to infections, inflammation, organ injury as well as after strenuous exercise.
The Neutrophil-Lymphocyte Ratio (NLR) is a significant biomarker used in live blood analysis to assess the balance between neutrophils and lymphocytes, two critical types of white blood cells involved in the body’s immune response. Neutrophils are the first responders to infection or injury, playing a key role in the inflammatory response, while lymphocytes are responsible for adaptive immunity, including the recognition of pathogens and immune memory.
Healthy, recoverable, Roulleux red cell formations. Cells are hibernating.
Always locate and photograph the best quality sample for this analysis.
Blue light filter
Moderate fibrin activity. Almost no crystals formation occurring. Crystals tend to be formed from fibrin.
Also can indicate high dehydration, acidity.
Ameba, Possible parasite egg, Heavy Rolleux
This is more an indication of serum versus blood cells balance. However a healthy and vibrant person will produce stronger and sustained blood movement than will a person who is dehydrated, under-nourished, does not exercise, suffers from stress issues of all types.
In the event parasites are detected, or eosinophils are low or high, suggest to the patient to take the Deep Dive Service.
Symptoms of parasitic infections depend on where in your body you’re infected. Some common symptoms include:
String bacteria, that could easily be mistaken for a parasitic worm. Bacteria usually shows green in color. Can be compared to mucous strings from lungs.
Atmospheric carbon crystal, not from blood.
In live blood analysis, the presence of both fungal elements and bacteria in the blood can be observed as an indicator of infection or imbalance in the body’s microbiome. Fungus, such as Candida species, may appear as distinct forms like yeasts or hyphal structures, while bacteria can show up as clusters or individual organisms depending on the type.
Candida albicans. Notice the formation is similar to a bunch of grapes. Larger white cells are can be seen attacking the edge of the infestation. This person has healthier immune function than the one pictures above.
Must be differentiated from similar looking bacteria.
“Odd biologicals” is a term used in live blood analysis to refer to unusual or atypical structures observed in the blood that do not belong to the typical range of red blood cells, white blood cells, or platelets. For example, the presence of abnormal cell shapes or unexplained inclusions in the blood could point to issues with cell regeneration, genetic mutations, or other underlying conditions. “Odd biologicals” could also refer to artifacts introduced by external factors such as improper blood collection techniques or contamination during sample preparation.
Low to moderate toxicity crystal, probably carbon – the most common of blood solids contaminants. Unstained sample.
Notice that it is affected surrounding red cells, there appears to be an exchange of elements between them.
This was probably inhaled by the patient, which is of course immediately transferred to blood.
Triglycerides. Note the barbs or macrophylla emanating from the edges – which makes it stick to other cells or vessel walls.
Easily mistaken for fungus or bacteria. Sticking to red cells, but not consuming them. Fungus or bacteria would probably consume adjacent red cells, causing a bleached or depleted appearance.
Also note heavy fibrin activity, indicating dehydration, and possible crystals formation.
Highly toxic double crystaline structures.
On the left is carbon or other dark element, perhaps lead. On the right is a lighter element, such as aluminum or something more toxic. Could also be related to graphene hydroxide with graphene oxide.
The orbiting Burr cells also indicate high toxicity. The surrounding red cells are all highly affected.
Mottled serum on the lower right can indicate acidity.
This can indicate a loosened fragment of a cancer elsewhere in the body.
Glass fragment, from the edge of a slide, not from the patient.
Note that the fragment diffracts light, is crystalline in structure. And is it not affecting, nor harming the surrounding cells.
‘Nano Delivery Tube’ presumably delivering mRNA materials.
Notice the vapor trail exiting to the right. Visible because the tube landed partially in higher pressure serum and a lower pressure air bubble.
Graphene Oxide is a two-dimensional (2D) material composed of carbon atoms. Its bi-dimensional nature causes unique interactions with blood proteins and biological membranes that can lead to unusual effects like blood clotting and immune cell activation, when combined with mRNA, lipid nanoparticles, and more. and dendritic cell progeny serve three main functions in the immune system.
Dr. Andreas Noack is a German expert in graphene nano structures. He describes these nanoscale structures as “tiny razor blades”. Only one atom layer thick, they are relatively wide and long. Fortunately we have observed their consumption by white blood cells, known as lymphocytes, in people with intact immune systems, these structure do deteriorate over time in a healthy, immunities-intact body.
Graphene hydrogel or nano gel is a water-based three dimensional (3D) graphene hybrid that readily absorbs water and swells to large volumes. Graphene hydrogel nanoparticles are outstanding drug delivery systems, owing to their unique properties that combine the characteristics of high water content with a very small (nano)
size. They combine well with drugs, and most importantly carry lipid nanoparticles in even distribution, while enhancing the action of LNP targeting ligands.
None of these structures have ever been observed by us before April of 2021. In some cases they appear to be hybrids of graphene, in other cases strictly biological, perhaps from sources that have hybridized them.
The characteristic common to all graphene structures observed in blood is that they consume red cells, perhaps for their iron content, and in production of hybrid structures of various types and purpose.
These can be the beginnings of red cell clots or amyloid clots. We have proven that EDTA chelation therapy breaks up these clots and helps remove them from the body.
Urine color can give clues about your health and hydration. Here’s a simpler breakdown:
Clear to Light Yellow: Healthy and well-hydrated.
Dark Yellow: Slight dehydration; drink more water.
Brown or Tea-Colored: Could mean dehydration or liver problems; consult a doctor.
Red or Pink: Could be blood in urine (infection, stones) or from foods like beets.
Orange: May be from dehydration, certain medications, or foods like carrots.
Cloudy: Could be a sign of infection or dehydration.
Urine clarity refers to how clear or cloudy your urine looks. Clear urine is usually a sign of good hydration, meaning you’re drinking enough water. Cloudy urine might indicate that something is wrong, like an infection, dehydration, or even the presence of excess minerals or mucus.
Urine pH refers to how acidic or alkaline (basic) your urine is. It is measured on a scale from 0 to 14, with 7 being neutral.
Urine specific gravity is a measure of the concentration of particles in urine, reflecting the kidney’s ability to balance water and waste. It is typically measured on a scale ranging from 1.000 (completely dilute) to 1.030 (highly concentrated).
The presence of protein in urine, known as proteinuria, can be an indicator of various health conditions. Under normal circumstances, urine contains little to no protein because the kidneys filter out waste and retain essential substances like proteins.
Glucose in urine, also known as glycosuria, refers to the presence of glucose (sugar) in the urine. Under normal conditions, the kidneys filter glucose from the blood and reabsorb it, preventing its loss in urine. However, when blood glucose levels are too high, the kidneys may not be able to reabsorb all of the glucose, resulting in its appearance in the urine.
Ketones in urine refer to the presence of ketone bodies, which are produced when the body breaks down fat for energy instead of carbohydrates. This usually happens when there is a lack of glucose, such as during fasting, a low-carbohydrate diet, or uncontrolled diabetes. The presence of ketones in urine can be detected through a test and may indicate conditions like diabetes, starvation, or a low-carb diet. High levels of ketones can be a sign of a medical issue and may require attention.
Nitrites in urine refer to the presence of nitrite compounds, which are typically produced when bacteria in the urinary tract convert nitrates into nitrites. The presence of nitrites in urine often indicates a urinary tract infection (UTI), as certain bacteria that cause these infections can trigger this process. Detecting nitrites in urine through a test can help diagnose a UTI.
Urobilinogen is a substance formed in the intestines from the breakdown of bilirubin, which is produced when the liver processes red blood cells. It is normally present in small amounts in urine. Higher or lower levels of urobilinogen in the urine may indicate liver disease, hemolysis, or other health conditions. Testing for urobilinogen can help assess liver function and overall health.
Bilirubin in urine refers to the presence of bilirubin, a substance produced when the liver breaks down old red blood cells. Normally, bilirubin is processed by the liver and removed through the bile, not found in urine. Its presence in urine can indicate liver problems, such as liver disease or bile duct obstruction. Testing for bilirubin in urine helps assess liver function and identify potential health issues.
Erythrocytes (Red Blood Cells) in urine refer to the presence of red blood cells in the urine, which is not normal. Their presence can indicate various health conditions, such as urinary tract infections, kidney stones, or injury to the urinary tract. A urine test can detect red blood cells, and their presence may require further investigation to determine the underlying cause.
Leukocytes (White Blood Cells) in urine refer to the presence of white blood cells in the urine, which can indicate an infection or inflammation in the urinary tract. Normally, white blood cells are part of the immune system and help fight infections. Their presence in urine is often a sign of conditions such as urinary tract infections (UTIs) or kidney disease. A urine test can detect leukocytes and help diagnose underlying health issues.
Dormancy Period: Bug can be dormant up to 12 months. No vectored diseases are known.
Although they move away from the host after feeding, they remain within the confines of their host’s roost, nest or dwelling. They may be considered to be micro-predatory bloodsuckers. Adult bedbugs have been reported to live three to twelve months if in an untreated household situation. The effects of cimicid feeding on the host include causing an immune response that results in discomfort, the transmission of pathogens, secondary infections at the wound site, physiological changes such as iron deficiency, and reduced fitness. Although viruses and other pathogens can be acquired by cimicids, they rarely transmit them to their hosts, unless the host is immune compromised.
Dormancy Period: From vectored pathogens up to 20 days.
Body lice may lay eggs on the host hairs and clothing, but clothing is where the majority of eggs are usually secured. The most important pathogens which are transmitted by them are Rickettsia prowazekii (causes epidemic typhus), Borrelia recurrentis (causes relapsing fever), and Bartonella quintana (causes trench fever). Adult lice can live for about thirty days, but if they are separated from their host they will die within two days.
Dormancy Period: Scrub Typhus, 21 days
Leptotrombidium deliense is considered a dangerous pest in East Asia and the South Pacific because it often carries Orientia tsutsugamushi, the tiny bacterium that causes scrub typhus, which is known alternatively as the Japanese river disease, scrub disease, or tsutsugamushi. The mites are infected by the Rickettsia passed down from parent to offspring before eggs are laid in a process called transovarial transmission. Symptoms of scrub typhus in humans include fever, headache, muscle pain, cough, and gastrointestinal symptoms.
Dormancy Period: Adult louse live for up to 30 days. No vectored diseases are known.
Feeding exclusively on blood, the crab louse usually is found in the person’s pubic hair. Although the louse cannot jump, it can also live in other areas of the body that are covered with coarse hair, such as the peri-anal area, the entire body (in men), and the eyelashes (in children).
The total life cycle from egg to adult is 16–25 days. Adults live for up to 30 days. Crab lice feed exclusively on blood, and take a blood meal 4–5 times daily. Outside the host they can survive for 24–48 hours. Crab lice are transmitted from person to person most commonly via sexual contact, although fomites (bedding, clothing) may play a minor role in their transmission. Crab lice are not known to transmit disease; however, secondary bacterial infection can occur from scratching of the skin. Symptoms of crab louse infestation in the pubic area include itching, redness and inflammation.
Dormancy Period: The total lifespan of a Demodex mite is several weeks, with skin diseases evolving over days or months.
Demodex canis lives on the domestic dog, can become mange, and are easily transferred from them. Demodicosis is most often seen in folliculitis (inflammation of the hair follicles of the skin). It may result in small pustules (pimples) at the base of a hair shaft on inflamed, congested skin. Demodicosis may also cause itching, swelling, and erythema of the eyelid margins. Scales at the base of the eyelashes may develop. Typically, patients complain of eyestrain. Older people are much more likely to carry face mites; about a third of children and young adults, half of adults, and two-thirds of elderly people carry them. The lower rate in children may be because children produce less sebum, or simply have had less time to acquire the mite. The six-legged larvae hatch after 3–4 days, and the larvae develop into adults in about 7 days. The total lifespan of a Demodex mite is several weeks.
Demodex mites are involved in psoriasis, allergic rhinitis, and seborrheic dermatitis in immuno-suppressed individuals. a correlation between Demodex infestation and acne vulgaris exists, suggesting it may play a role in promoting acne, including in immunocompetent infants displaying pityriasis and erythema toxicum neonatorum. Studies suggest an association between mite infestation and rosacea.
Dormancy Period: Several months without food. Numerous dangerous vectors can emerge up to years later.
Fleas feed on a wide variety of warm-blooded vertebrates including dogs, cats, rabbits, squirrels, ferrets, rats, mice, birds, and sometimes humans. Female fleas can lay 5000 or more eggs over their life, an adult flea only lives for 2 or 3 months. Without a host to provide a blood meal. A flea’s life can be as short as a few days, or can live for up to a year and a half, can live for several months without eating, so long as they do not emerge from their puparia.
Dormancy Period: Adult lice will die within 2 days without a blood meal. Rare vectors in Africa with up to 20 days incubation.
Head lice feed only on human blood and are only able to survive on human head hair. They only spread by human to human contact. When adults, they are about 2 to 3 mm long. When not attached to a human, they are unable to live beyond three days. In Ethiopia, head lice appear to be able to spread louse-born epidemic typhus and Bartonella quintana. Elsewhere head lice do not appear to carry these infections.
Dormancy Period: Rickettsialpox is generally mild and resolves within 2–3 weeks if untreated. There are no known deaths resulting from the disease. Other vectors have been lab tested but not proven outside the lab.
It can transmit human disease, is associated with causing rodent mite dermatitis in humans and is noted for carrying Rickettsia akari, which causes rickettsialpox. Rodent mites are capable of surviving for long periods without feeding and traveling long distances when seeking hosts. Cases have been reported in homes, libraries, hospitals and care homes. A similar condition, known as gamasoidosis, is caused by avian mites.
Dormancy Period: Up to several years with vectored diseases.
Mosquito-borne diseases or illnesses are caused by bacteria, viruses, or parasites transmitted by mosquitoes. Nearly 700 million people contract mosquito-borne illnesses each year, resulting in more than a million deaths.
Diseases transmitted by mosquitoes include malaria, dengue, West Nile virus, chikungunya, yellow fever, filariasis, tularemia, dirofilariasis, Japanese encephalitis, Saint Louis encephalitis, Western equine encephalitis, Eastern equine encephalitis, Venezuelan equine encephalitis, Ross River fever, Barmah Forest fever, La Crosse encephalitis, and Zika fever, as well as newly detected Keystone virus and Rift Valley fever.
Dormancy Period: Up to many years depending on the related vector.
Diagnosis can be challenging as the small size of avian mites make them “barely visible to the unaided eye”. Dermanyssus gallinae can also infest various body parts, including the ear canal and scalp. commonly found in the bedroom or where the patient sleeps, as they prefer to stay close to their host for optimal feeding. D. gallinae generally visit their host for up to 1–2 hours, leave after completing their blood meal, and typically feed every 2–4 days. They are able to move extremely quickly, and can take less than 1 second to bite; enough time to inject their saliva and to induce rash and itching.They locate potential hosts through temperature changes, vibrations, chemical signals and CO2.
Dormancy Period: Up to six weeks.
Scabies, also sometimes known as the seven-year itch, is a contagious human skin infestation by the tiny (0.2–0.45 mm) mite Sarcoptes scabiei, In a first-ever infection, the infected person usually develops symptoms within two to six weeks. During a second infection, symptoms may begin within 24 hours. The mites burrow into the skin to live and deposit eggs.The symptoms of scabies are due to an allergic reaction to the mites. Scabies is most often spread during a relatively long period of direct skin contact with an infected person (at least 10 minutes) such as that which may occur during sexual activity or living together. Spread of the disease may occur even if the person has not developed symptoms yet.
Dormancy Period: Up to several years, depending on the vector.
Ticks are external parasites, living by feeding on the blood of mammals, birds, and sometimes reptiles and amphibians. Ticks have up to seven nymphal stages (instars), each one requiring blood ingestion, and as such, Ticks undergo a multihost life cycle. Because of their hematophagous (blood-ingesting) diets, ticks act as vectors of many serious diseases that affect humans and other animals.
Dormancy Period: May remain undetected for many years, however anemia may be an indicator of long term infection..
Hookworms account for a high proportion of debilitating disease in the tropics and 50–60,000 deaths per year These worms produce an iron deficiency anemia by sucking blood from the host’s intestinal walls.
Dormancy Period: The incubation period in humans is usually from 1 week to 47 days after infection. Most cases are asymptomatic.
In humans, A. cantonensis is the most common cause of eosinophilic meningitis or meningoencephalitis. Frequently the infection will resolve without treatment or serious consequences, but in cases with a heavy load of parasites the infection can be so severe it can cause permanent damage to the central nervous system or death.
Dormancy Period: If no immediate allergic reaction, more severe digestive reactions may be experienced within a few days.
Anisakiasis is a human parasitic infection of the gastrointestinal tract caused by the consumption of raw or undercooked seafood containing larvae of the nematode Anisakis simplex. Reactions, mostly seen as fish allergies, tend to occur soon after consumption.
Dormancy Period: Up to 3 years.
Often, people show no overt symptoms but may suffer from intestinal problems. When symptoms do occur, the person is usually infected with a large number of worms. Ascaris lumbricoides is one of the most difficult pathogens to kill (second only to prions), and the eggs commonly survive 1–3 years before hatching.
Dormancy Period: Several years asymptomatic.
Most people are asymptomatic unless heavily infected. Human infection with Baylisascaris procyonis has been relatively rare. However, disease caused by this parasite can be extremely dangerous, causing death or severe symptoms. The parasite has been known to infect more than 90 kinds of wild and domestic animals. Reported disease has primarily afflicted children and almost all cases were a result of the ingestion of contaminated soil or feces, via the oral fecal route. the infection results in the penetration of the gut wall by the larvae and subsequent invasion of tissue, resulting in severe disease.
Dormancy Period: Up to 50 days.
After maturing for approximately 50 days, the juveniles then migrate to the kidneys (typically the right kidney). Upon maturation, D. renale can survive for five years. D. renale is distributed worldwide, but is less common in Africa and Oceania. It affects fish-eating mammals, particularly mink, wolves, coyotes, foxes, dogs, raccoons, and weasels. Human infestation is rare, but results in kidney destruction.
Dormancy Period: Possibly days to years.
Ophidascaris robertsi is a nematode (also known as roundworm) usually parasitic in the carpet python (Morelia spilota). It is found in Australia and Papua New Guinea,and possibly Indonesia. Pythons serve as the typical hosts for Ophidascaris robertsi. Humans and mammals that live near carpet python habitat and forage for native vegetation to cook can be exposed by consuming the roundworm’s eggs.These eggs, which are commonly shed in snake droppings due to the snakes’ diet of infected animals, likely contaminates the grass and soil eaten by small mammals. Other vectors, such as domestic and wild animals, are yet to be investigated.
Dormancy Period: One year or longer. The first signs of dracunculiasis occur around a year after infection, as the full-grown female worm prepares to leave the infected person’s body.
About a year after the initial infection, the female migrates to the skin, forms an ulcer, and emerges. When the wound touches fresh water, the female spews a milky-white substance containing hundreds of thousands of larvae into the water.
Dormancy Period: Up to 8 weeks, often asymtomatic.
The disease is spread between people by pinworm eggs. The eggs initially occur around the anus. The period of time from swallowing eggs to the appearance of new eggs around the anus is 4 to 8 weeks. The main symptoms are itching in and around the anus and perineum. One-third of individuals with pinworm infection are totally asymptomatic. The eggs are hardy and can remain infectious, outside the body, in a moist environment for up to three weeks.
Dormancy Period: Up to 4 weeks.
Gnathostomiasis is transmitted by the ingestion of third-stage larvae from raw or insufficiently cooked second intermediate or paratenic hosts such as freshwater fish, snakes, poultry, or frogs. The incubation period for gnathostomiasis is 3–4 weeks when the larvae begin to migrate through the subcutaneous tissue of the body.
Dormancy Period: Possibly days to weeks.
Halicephalobus gingivalis is a free-living saprophagous nematode species. It is a facultative parasite of horses, invading the nasal cavity, and sometimes numerous other areas, where it produces granulomatous masses. On rare occasion, it can infect humans as well, causes a universally lethal meningoencephalitis. Infection of the brain is common, followed by the kidneys, oral and nasal cavities, lymph nodes, lungs, spinal cord, and adrenal gland, and also reports of infection of heart, liver, stomach and bone.
Dormancy Period: Up to 1 year.
Loa loa filariasis, (Loiasis) is a skin and eye disease caused by the nematode worm Loa loa. Humans contract this disease through the bite of a deer fly (Chrysops spp.) or mango fly. These carriers are blood-sucking and day-biting, and they are found in rainforest-like environments in western and central Africa.
Dormancy Period: Days to weeks
The infection of these roundworms typically causes no overt symptoms but may sometimes cause a mild dermatitis of the thorax and shoulders. M. streptocerca infections fortunately do not cause any nodules, skin disease, or ocular infections like that of Onchocerca volvulus. However they may become visible just under the skin surface, and perhaps decrease skin health and immunity.
Dormancy Period: 12 months to 15 years.
The average adult worm lifespan is 15 years, and mature females can produce between 500 and 1,500 microfilariae per day. The normal microfilarial lifespan is 1.0 to 1.5 years; however, their presence in the bloodstream causes little to no immune response until death or degradation of the microfilariae or adult worms. It is spread from person to person via female biting blackflies of the genus Simulium, and humans are the only known definitive host.
Dormancy Period: Lifetime of the patient.
The adult parasitic stage lives in tunnels in the mucosa of the small intestine. Many people infected are asymptomatic at first. Symptoms include dermatitis: swelling, itching, larva currens, and mild hemorrhage at the site where the skin has been penetrated. Spontaneous scratch-like lesions may be seen on the face or elsewhere.
Dormancy Period: Days to 1 year.
Thelaziasis is the term for infestation with parasitic nematodes of the genus Thelazia. The adults of all Thelazia species discovered so far inhabit the eyes and associated tissues (such as eyelids, tear ducts, etc.) of various mammal and bird hosts, including humans. Thelazia nematodes are often referred to as “eyeworms”.
Dormancy Period: 2 weeks to several years.
Toxocariasis is an illness of humans caused by the dog roundworm (Toxocara canis) and, less frequently, the cat roundworm (Toxocara cati). These are the most common intestinal roundworms of dogs, coyotes, wolves and foxes and domestic cats. Humans are among the many “accidental” or paratenic hosts of these roundworms.
Dormancy Period: Up to 7 days.
About 11 million humans are infected with Trichinella. The great majority of trichinosis infections have either minor or no symptoms and no complications. Trichinosis. During the initial infection, invasion of the intestines can result in diarrhea, abdominal pain, and vomiting. Migration of larvae to muscle, which occurs about a week after being infected, can cause swelling of the face, inflammation of the whites of the eyes, fever, muscle pains, and a rash. Complications may include inflammation of heart muscle, central nervous system involvement, and inflammation of the lungs.
Dormancy Period: 3 months, up to 1 year or longer.
For about four weeks, the whipworms feed on blood vessels located within the cecum of the large intestine. Eventually, the whipworms leave the cecum and begin to lay thousands of eggs. These unembryonated eggs are then released from the host through feces. The process from egg ingestion to release takes around 12 weeks. The released eggs become embryonated in approximately nine to twenty-one days and are eventually ingested by another host. Eggs that are passed in the feces, can remain alive in soil for years.
Dormancy Period: a few days to a few weeks, but usually it is about two to four weeks.
Most infected people, about 90%, are asymptomatic, but this disease has the potential to become serious. It is estimated that about 40,000 to 100,000 people worldwide die annually due to amoebiasis
Since amoebiasis is transmitted through contaminated food and water, it is often endemic in regions of the world with limited modern sanitation systems, including México, Central America, western South America, South and Southeast Asia, and western and southern Africa.
Dormancy Period: a few days to the lifetime of the patient.
Balantidiasis is a zoonotic disease and is acquired by humans via the feco-oral route from the normal host, the pig, where it is asymptomatic. Fecally contaminated food and water are the common sources of infection in humans.
Dormancy Period: weeks to years.
Blastocystis is a protozoal, single-celled parasite that inhabits the gastrointestinal tracts of humans and other animals. Many different types of Blastocystis exist, and they can infect humans, farm animals, birds, rodents, amphibians, reptiles, fish, and even cockroaches. Blastocystosis has been found to be a possible risk factor for development of irritable bowel syndrome.
Dormancy Period: 2- 28 days.
Cryptosporidiosis, sometimes informally called crypto, is a parasitic disease caused by Cryptosporidium, a genus of protozoan parasites in the phylum Apicomplexa. It affects the distal small intestine and can affect the respiratory tract in both immunocompetent (i.e., individuals with a normal functioning immune system) and immunocompromised (e.g., persons with HIV/AIDS or autoimmune disorders) individuals, resulting in watery diarrhea with or without an unexplained cough. In immunosuppressed individuals, the symptoms are particularly severe and can be fatal. It is primarily spread through the fecal-oral route, often through contaminated water; recent evidence suggests that it can also be transmitted via fomites contaminated with respiratory secretions.
Incubation Period: 1 week
Cyclosporiasis primarily affects humans and other primates. When an oocyst of Cyclospora cayetanensis enters the small intestine, it invades the mucosa, where it incubates for about one week. After incubation, the infected person begins to experience severe watery diarrhea, bloating, fever, stomach cramps, and muscle aches.
Incubation Period: Days.
Dientamoebiasis is a medical condition caused by infection with Dientamoeba fragilis, a single-cell parasite that infects the lower gastrointestinal tract of humans. It is an important cause of traveler’s diarrhea, chronic abdominal pain, chronic fatigue, and failure to thrive in children.
Dormancy Period: Weeks to months.
Leishmaniasis is a wide array of clinical manifestations caused by protozoal parasites of the Trypanosomatida genus Leishmania. It is generally spread through the bite of phlebotomine sandflies, Phlebotomus and Lutzomyia, and occurs most frequently in the tropics and sub-tropics of Africa, Asia, the Americas, and southern Europe.
Dormancy Period: Up to 12 days, death up to two weeks after exposure. Early and accurate diagnosis is essential.
Naegleria fowleri, also known as the brain-eating amoeba. This free-living microorganism primarily feeds on bacteria but can become pathogenic in humans, causing an extremely rare, sudden, severe, and usually fatal brain infection known as naegleriasis or primary amoebic meningoencephalitis (PAM).
Feces-Borne Endoparasites and Pathogens
Dormancy Period: Up to 9 weeks, or the life of the patient if asymptomatic.
People can get infected with Babesia parasites by the bite of an infected tick, by getting a blood transfusion from an infected donor of blood products, or by congenital transmission (an infected mother to her baby). Ticks transmit the human strain of babesiosis, so it often presents with other tick-borne illnesses such as Lyme disease.
Dormancy Period: 2 months up to several years.
An estimated 6 to 7 million people worldwide are infected with T. cruzi Chagas disease. Chagas disease is caused by infection with the protozoan parasite T. cruzi, which is typically introduced into humans through the bite of triatomine bugs, also called “kissing bugs”.
Dormancy Period: Days to weeks.
Balamuthia mandrillaris is a free-living amoeba that causes the rare but deadly neurological condition granulomatous amoebic encephalitis (GAE). B. mandrillaris can infect the body through open wounds or possibly by inhalation. It is distributed throughout the temperate regions of the world.
Dormancy Period: 1 week to months.
Acanthamoeba spp. are among the most prevalent protozoa found in the environment. They are distributed worldwide, and have been isolated from soil, air, sewage, seawater, chlorinated swimming pools, domestic tap water, bottled water, dental treatment units, hospitals, air-conditioning units, and contact lens cases. Additionally, they have been isolated from human skin, nasal cavities, throats, and intestines, as well as plants and other mammals.
Dormancy Period: Up to 24 weeks after initial symptoms.
Human malaria is caused by single-celled microorganisms of the Plasmodium group. It is spread exclusively through bites of infected female Anopheles mosquitoes. The mosquito bite introduces the parasites from the mosquito’s saliva into a person’s blood. The parasites travel to the liver, where they mature and reproduce.
Dormancy Period: Can remain a dormant fungus for years.
This organism infects the mucosa of the nasal cavity, producing a mass-like lesion. This mass appears to be polypoidal in nature with a granular surface speckled with whitish spores. The rhinosporidial mass has been classically described as a strawberry-like mulberry mass. This mass may extend from the nasal cavity into the nasopharynx and present itself in the oral cavity. These lesions commonly cause bleeding from the nasal cavity. R. seeberi can also affect the lacrimal gland and also rarely the skin and genitalia.
Dormancy Period: Up to 60 days. Many are asymptomatic.
If symptoms develop, they typically occur 20–40 days after ingestion of sporocysts and during the subsequent migration of sporozoites through the body vessels. Acute lesions (edema, hemorrhages, and necrosis) develop in the affected tissues. The parasite has a predilection for skeletal muscle (myositis), cardiac muscle (petechial hemorrhages of cardiac muscle and serosae), and lymph nodes (edema, necrosis, and hemorrhage).
Dormancy Period: Unknown.
T. brucei is transmitted between mammal hosts by an insect vector belonging to different species of tsetse fly (Glossina). Transmission occurs by biting during the insect’s blood meal. Trypanosoma brucei is a species of parasitic kinetoplastid belonging to the genus Trypanosoma that is present in sub-Saharan Africa.
Dormancy Period: 7-10 days, or in dormancy the lifetime of the patient.
Found worldwide, T. gondii is capable of infecting virtually all warm-blooded animals. In humans, particularly infants and those with weakened immunity, T. gondii infection is generally asymptomatic but may lead to a serious case of toxoplasmosis. T. gondii can initially cause mild, flu-like symptoms in the first few weeks following exposure, but otherwise, healthy human adults are asymptomatic.
Dormancy Period: Several years in asymptomatic patients.
Trichomonas is a genus of anaerobic excavate parasites, and is estimated to be the most prevalent non-viral STI worldwide. Infection rates in men and women are similar but women are usually symptomatic, while infections in men are usually asymptomatic. Transmission usually occurs via direct, skin-to-skin contact with an infected individual, most often through vaginal intercourse. 160 million cases of infection are acquired annually worldwide.
The world has changed faster than the body can adapt. Every moment of every day sees unfathomable mixtures of chemicals, vehicle exhausts, food additives and preservatives, medical vaccinations, antibiotics, pharmaceuticals, and stresses of every kind assaulting the delicate balance that is human life. The liver filters and stores much of these stressors, but cannot always process them. When the liver becomes over-burdened, skin blemishes, aching joints, gas, bloating, weight gain, fatigue, and myriad other reactions often result.
Detox & prevention are the core of services offered by BSI. We are science-based in terms of diagnosis and treatment, however the medicines are natural and one-of-a-kind by Therapure Nutraceuticals, creating a targeted, more accurate, immune-boosting, natural healing process. We examine and explore patient’s overall health, designing a customized plan to cleanse the body from many of the environmental and conditioned toxins that make their way into our everyday lives.
BSI International Clinics views disease and its causes very differently, a methodology rarely known to most patients. An holistic science-based approach (discerning causes of disease through scientific procedure, and healing with appropriate natural medicines and techniques) means we get to know you, spending approximately two or more hours together over two visits.
And during the first visit, many patients also request a standard intravenous infusion, type and dose based on answers from the questionnaire. This IV may include 1 or 3 grams (1,000 or 3,000 mg) of vitamin C / sodium ascorbate, vitamins B1, B6, and B12, magnesium, EDTA chelate, electrolytes, all in 500 ml of pure water, taken over 90 minutes or less.
All new Member Patients are requested to complete the Health History Questionnaire and Full Basic Testing.
Tel: +62 812 3838 4040
medical@bsi.international
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